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Hepatic cirrhosis

Liver cirrhosis is the ultimate consequence of many liver diseases and chronic poisoning. Cirrhosis is usually associated with the fact that chronic liver disease causes chronic hepatitis (hepatitis) , which causes continuous necrosis and hepatic lobe regeneration to cause liver cirrhosis (cirrhosis). Cirrhosis is the result of multiple liver damage and healing. Cirrhosis is a strong risk factor for liver cancer (hepatoma) .

Cirrhosis of the liver

The regenerative capacity of the liver is high. Even in normal operation, the liver lobes function as a filter of blood coming from the intestines and the body and, like the oil filter in the car, require more frequent replacement. Clogged and damaged liver lice die and are replaced by new ones. However, as with any healing, liver regeneration produces scar tissue (fibrous). If the liver is overloaded with chronic infection or intoxication, the rate of scar tissue formation is much higher. Heavily loaded liver gradually builds up so much collagen that by his own volume prevents the regeneration of new liver beams. The liver is as hard as a sinewy snout - that's why it is called cirrhosis ( hardening of the liver ).

Consequences of cirrhosis

Once cirrhosis develops, most of the liver volume is made up of unnecessary collagen tissue. The remaining useful liver volume is insufficient for the need to filter the blood returning the portal vein from the intestines and the gastrointestinal tract. The situation is the same as for each filter - because of reduced throughput, venous pressure increases in all parts of the digestive tract, which include the esophagus, the stomach, the small intestine, and the large intestine. In particular, the esophagus is prone to so-called varicose esophageal varicose veins - a diseased extension of the livers of the esophagus. Circulatory liver is not enough to purify blood, LDL cholesterol increases in blood and accumulates waste particles. This contributes to the rapid development of atherosclerosis . In addition, Cirhotic Liver tries to regenerate new beams, but there is no room for it. This leads to continuous attempts of liver cells (in which mutations are already accumulated due to previous inflammations and poisoning) to divide and susceptibility of a patient with cirrhosis to primary liver cancer (hepatoma). In summary, cirrhosis of the liver creates a syndrome of mutually reinforcing pathological tendencies that significantly shorten the life of the affected person.

Protection against cirrhosis

Once cirrhosis develops, it is incurable. If liver transplantation is over, it is best to protect our liver from chronic inflammation by hygiene, healthy lifestyle and hepatoprotective adaptogens. The existence of hepatoprotective (liver-protecting) plants and fungi is real, but it is not entirely clear what all these are and how their effects are compared to each other. In this situation, I recommend that you rely on the tradition of TČM and maybe also Ayurveda, to which we add at least of our herbs at least a millet thistle .

Otherwise, the herb protecting the herb includes both the model plant ginseng adaptogen and the gloss gloss glossy model adaptation sponge . The hepatoprotective effect of ginseng is not significant. According to study Lo2011gri , ginsenoside ginsenoside Rb 1 acts against the formation of fibrotic tissue in the liver, and according to Tark2015egr , this ginsenoside (Rb 1 ) also has a potential positive effect on remodeling of scar tissue, which gives some hope to patients with already developed cirrhosis. The antifibrotic effect of gloss glossy in the liver is documented in the Kwon2011aaf and Sudheesh2012glp studies . You will find a list of other liver-protecting adapters under the motto of liver disease .

| 25.7.2008